Acute Acute Tubular Necrosis
Acute tubular necrosis is damage to the tubule cells in the kidney that results in kidney failure. It result when tubular cells do not get enough oxygen or when they have been exposed to a toxic drug or molecule. The role of reactive oxygen molecules has been further elucidated, and novel antioxidants and iron chelators have been identified. The tubular cells of the kidneys undergo a continuous cycle of cell death and renewal, much like the cells of the skin. Acute tubular necrosisis caused by ischemia of the kidneys and by exposure to materials that are poisonous to the kidneys. Risks for acute tubular necrosis include injury or trauma with resulting damage to the muscles, recent major surgery and postischemic. Acute-tubuler-necrosis is characterized by a rising plasma creatinine concentration, a urine volume that may be reduced or normal, and a characteristic set of changes in the urinalysis, including many granular casts and a fractional excretion of sodium above one percent. There was no difference in the incidence of acute tubular necrosis between splenectomized and nonsplenectomized patients who received cadaveric grafts or kidneys from living related donors.
Causes of Acute Acute Tubular Necrosis
The common Causes of Acute Acute Tubular Necrosis
- Low blood pressure.
- Infection in the blood .
- Yellow-tinged skin.
- Dye used for radiographic studies
- cisplatin.
- Herbal and folk remedies.
- Exposure to toxic materials.
Symptoms of Acute Acute Tubular Necrosis
The common Symptoms of Acute Acute Tubular Necrosis
- vomiting.
- Coma.
- Hard to arouse.
- Nausea.
- Change in urine output.
- Fluid retention.
- Chills shaking.
Treatment of Acute Acute Tubular Necrosis
The common Treatment of Acute Acute Tubular Necrosis
- Dialysis.
- Diuretics.
- Certain medications eg.Mannitol, Fenoldpam and Auriculin anaritide.
- Transplantation of kidneys.
|
